How can genomics help with understanding and treating this condition?
THC binding to 5-HT2A may activate signaling pathways that cause negative effects on cognition and memory.
Stronger binding interactions between MDMB-Fubinaca and CB1 may explain why some synthetic cannabinoids cause dangerous effects.
Tetrahydrocannabiphorol could account for the pharmacological properties of some cannabis varieties which are difficult to explain by the presence of THC alone.
Yes, and it may work via 5-HT2A as well as the cannabinoid receptors.